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FITC標(biāo)記(jì)的醛(quán)縮酶2抗體(tǐ)

文(wén)字:[大][中][小(xiǎo)] 2017-4-28    瀏覽(lǎn)次(cì)數:1686    

                                                       FITC標記的醛(quán)縮酶2抗體                                                                                                                                                
英文(wén)名(míng)稱(chēng)Anti-ALDOB/FITC
中文名稱(chēng):FITC標記的(dí)醛(quán)縮酶2抗體(tǐ)
別    名(míng)ALDB; ALDO B; ALDO2; ALDOB; ALDOB_HUMAN; Aldolase 2; Aldolase B; Aldolase B fructose bisphosphate; Aldolase2; AldolaseB; EC 4.1.2.13; Fructose bisphosphate aldolase B; Fructose-bisphosphate aldolase B; Liver type aldolase; Liver-type aldolase; MS1077.  

詳(xiáng)細(xì)介(jiè)紹:


規(guī)格(gé):100ul 
說 明(míng) 書100ul  
研究(jiū)領(lǐng)域腫瘤  細胞生(shēng)物(wù)  免疫(yì)學(xué)  信(xìn)號(hào)轉(zhuǎn)導  轉錄調節因子  
抗(kàng)體來(lái)源Rabbit
克隆類(lèi)型(xíng)Polyclonal
交叉反應(yīng) Human, Mouse, Rat, Pig, Cow, Rabbit, Sheep, 
產品(pǐn)應(yīng)用
not yet tested in other applications.
optimal dilutions/concentrations should be determined by the end user.
分 子(zǐ) 量39kDa
性    狀(zhuàng)Lyophilized or Liquid
濃    度(dù)1mg/ml
免(miǎn) 疫 原(yuán)KLH conjugated synthetic peptide derived from human ALDOB
亞(yà)    型IgG
純(chún)化(huà)方法affinity purified by Protein A
儲 存(cún) 液0.01M TBS(pH7.4) with 1% BSA, 0.03% Proclin300 and 50% Glycerol.
保存條(tiáo)件(jiàn)Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

相關(guān)資料:


產(chǎn)品介(jiè)紹(shào)background:
ALDOB is a tetrameric glycolytic enzyme that catalyzes the reversible conversion of fructose-1,6-bisphosphate to glyceraldehyde 3-phosphate and dihydroxyacetone phosphate. Defects in ALDOB cause hereditary fructose intolerance.

Subunit:
Homotetramer.

DISEASE:
Hereditary fructose intolerance (HFI) [MIM:229600]: Autosomal recessive disease that results in an inability to metabolize fructose and related sugars. Complete exclusion of fructose results in dramatic recovery; however, if not treated properly, HFI subjects suffer episodes of hypoglycemia, general ill condition, and risk of death the remainder of life. Note=The disease is caused by mutations affecting the gene represented in this entry.

Similarity:
Belongs to the class I fructose-bisphosphate aldolase family.

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