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今天是(shì)2026年7月22日 星期(qī)三,歡(huān)迎(yíng)光臨本站 上海(hǎi)研(yán)生實(shí)業(yè)有(yǒu)限(xiàn)公(gōng)司 網址(zhǐ): darylliu.cn

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FITC標記的酰(xiān)基(jī)輔(fǔ)酶A脫氫酶中鏈抗體

文字(zì):[大][中(zhōng)][小] 2017-5-3    瀏覽次數(shù):1481    

                                   FITC標(biāo)記(jì)的酰基(jī)輔(fǔ)酶A脫(tuō)氫酶中(zhōng)鏈抗體(tǐ)                                                                                                                                                
英(yīng)文名(míng)稱Anti-ACADM/FITC
中文名稱(chēng):FITC標(biāo)記(jì)的酰(xiān)基輔(fǔ)酶(méi)A脫氫酶中鏈(liàn)抗(kàng)體(tǐ)
別    名mitochondrial antibody; ACAD 1; ACAD1; Acyl coenzyme A dehydrogenase; Acyl coenzyme A dehydrogenase C 4 to C 12 straight chain; MCAD; MCADH; Medium chain acyl CoA dehydrogenase; Medium chain fatty acyl CoA dehydrogenase; Medium chain specific acyl CoA dehydrogenase; Medium chain specific acyl CoA dehydrogenase mitochondrial; FLJ18227; FLJ93013; FLJ99884; ACADM_HUMAN.   

詳(xiáng)細介(jiè)紹:


規格:100ul 
說(shuō) 明(míng) 書100ul  
研究領域腫(zhǒng)瘤  細胞(bāo)生(shēng)物  免(miǎn)疫學  轉錄調節(jié)因子  綫(xiàn)粒體(tǐ)  
抗(kàng)體來源Rabbit
克(kè)隆(lóng)類(lèi)型Polyclonal
交叉(chā)反應 Human, Mouse, Rat, Chicken, Dog, Pig, Cow, Horse, Rabbit, 
產品應用IF=1:50-200  
not yet tested in other applications.
optimal dilutions/concentrations should be determined by the end user.
分 子 量47kDa
性(xìng)    狀(zhuàng)Lyophilized or Liquid
濃    度1mg/ml
免(miǎn) 疫(yì) 原KLH conjugated synthetic peptide derived from human ACADM
亞    型IgG
純(chún)化(huà)方法(fǎ)affinity purified by Protein A
儲 存(cún) 液0.01M TBS(pH7.4) with 1% BSA, 0.03% Proclin300 and 50% Glycerol.
保存條(tiáo)件Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

相(xiāng)關資料(liào):


產品介紹background:
ACADM protein is a medium chain specific (C4 to C12 straight chain) acyl Coenzyme A dehydrogenase. The enzyme catalyzes the initial step of the mitochondrial fatty acid beta oxidation pathway. ACADM expression is induced during periods of fasting, when reliance on fatty acids for energy is increased. Clinical phenotypes are associated with ACADM hereditary deficiency.


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